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Parte 1: Epidemiología, fisiopatología y clínica. Seguimiento neumológico de los niños con displasia broncopulmonar al alta de la Unidad de Cuidados. Epidemia de displasia broncopulmonar: incidencia y factores asociados en una cohorte de niños prematuros en Bogotá, Colombia. Juan G. Ruiz-Peláez1,2,3. Displasia Broncopulmonar. ES. eliana silva. Updated 6 September Transcript. Displasia Broncopulmonar Diagnostico general. Nesecidad de mantener.

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Tissue microarrays were collected from lung samples from all the cases and analyzed immunohistochemically four samples for each case with 3 mm diameter eachsince this is the most suitable technique to analyze protein expression in this type of material 17 Fas is a protein that belongs to the subgroup of tumor necrosis factor receptor TNF-Rthis protein can trigger apoptosis.

In the literature, the function of Caspase 3 is not clear, Caspase 3 could be involved in the pathophisiology of oxygen and ventilation inducing apoptosis Enhancement and inhibition of apoptosis and cell proliferation play an important role in regulatory mechanisms involved in tissue remodeling 1.

Картинки: Displasia broncopulmonar fisiopatologia

Bienvenido a siicsalud Contacto Inquietudes. For each case, an average percentage of positive area was determined in 12 HPF images. No perinatal variable was associated with the above anemia, although we found that the prevalence of fisiopatolkgia disease decrease according the increase in gestational age at birth linear trend: Decreased alveolarization in baboon survivors with bronchopulmonary dysplasia.

The scores for each case were determined by adding the scores for each of the four samples A Management guideline to reduce the frequency of blood transfusion in very- low-birth-weight lq. PCNA is a nuclear protein which acts as an accessory factor of deoxyribonucleic acid DNA polymerase delta, it is required for DNA replication and repair, and consequently for cell replication Prematurity anemia, very early anemia, phlebotomies, anemia. The samples were then incubated overnight with the following primary antibodies: The presence of maternal pathology such as hypertensive disorders of pregnancy and gestational diabetes did not influence the incidence of CLD.

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The length of oxygen therapy use and the time of survival were almost a like.

It can allow recruiting Caspase 8 or Caspase 10 to the activated Fas receptor. Confirming these findings, previous studies in animal models have shown that there is increased apoptosis in lungs exposed to oxygen, which are susceptible to barotrauma, volutrauma and biotrauma 11 Randomized trial of liberal versus restrictive guidelines for red blood cell transfusins in pretern infants. However, the most important risk factor is the exposure to oxygen and mechanical ventilation 5.

The pathophysiology of bronchopulmonary dysplasia BPD can be the key for prevention of this disease, research with cell cycle proteins appear to be very promising for elucidating this pathology. The pattern of staining was scored as follows: Delayed broncopumonar clamping in the interest of the newborn child.

The medical records were analyzed to collect the data related to the clinical events such as gender, gestational age, birth weight, Apgar score at the first e fifth minute, pregnancy hypertension event, gestational diabetes, chorioamnionitis, longer time with amniotic sac disruption, asphyxia, antibiotic therapy, surfactant therapy, necrotizing enterocolitis, bronchopneumonia, pulmonary hemorrhage, pulmonary hypertension, intracranial hemorrhage, sepsis, corrected age postpartum and time spent on mechanical ventilation and oxygen therapy.

Lx 8 immunoexpression was also greater in the “new” CLD group. The authors state that they have obtained appropriate institutional review board approval or have followed the principles outlined in the Declaration of Helsinki for all human or animal experimental investigations.

Anti-Capase 3 is a rabbit polyclonal antibody, bromcopulmonar 3CSP03, 1: Peng H, Tong XM. Aberrant signaling pathways of the lung mesenchyme and their contributions to the pathogenesis of bronchopulmonary dysplasia.

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On displssia other hand, the “classic” group had more time for cell differentiation, a process that is characteristic for this phase. June 29, ; Accepted: For PTEN and Bcl-2, a score was assigned according to the degree of positive staining as well as a pattern of staining. East Mediterr Health J.

Displasia Broncopulmonar by eliana silva on Prezi

Anti-Caspase 8 is a rabbit polyclonal antibody, 1: Study sample included 25 neonates determining in each the presence or not of a very early anemia, as well as the prevalence of this disease and the associated maternal, perinatal and postnatal factors.

Es posible evitar las transfusiones de sangre en la anemia del prematuro. In recent years, the frequency of this disease has risen mainly because of the increased survival of very low birth weight preterm neonates with disruption of vascular and lung development linked to functional alterations related to surfactant deficiency and immaturity.

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These findings may be associated with the ability of a pulmonary cell to proliferate in response to mechanical strain 3334knowing that the “classic” form was submitted to mechanical ventilation for more days than the group “without” CLD. In the present study, Bcl-2 expression was higher in “classic” CLD than in the group “without” CLD Table 5but this finding has not been statistically significant.

The data were analyzed using SPSS The pathological features of the “classic” form showed an groncopulmonar bronchiolitis, an interstitial fibrosis that is compatible with proliferative activity.